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Covalent/irreversible inhibitors target the redox/catalytic apparatus of TGR with almost no recognition elements that give affinity and selectivity for TGR over off-targets and the cellular redox apparatus resulting in undesirable cross-reactions and low specificity in vivo 25

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[30] The main type of vitamin B 12 deficiency anemia is pernicious anemia, [31] characterized by a triad of symptoms: Anemia with bone marrow promegaloblastosis (megaloblastic anemia)

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